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Increased CaMKK2 activity in the brain leads to CaMK4 activation and increased expression of BDNF, which in mice is indispensable for the anti-manic actions of lithium [45, 49]

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S., Garzon, S., Casarin, J., Franchi, M., & Ghezzi, F

Subsequently, the normalized counts were transformed to CPM values and a log 2 transformation was applied using the R Bioconductor package edgeR (64)

More importantly, the dysfunction of Na + /K + -ATPase leads to secondary mitochondrial damage through ion imbalances: intracellular Na + accumulation promotes reverse operation of the sodium-calcium exchanger (NCX), resulting in intracellular calcium overload (85), which is a key potential trigger for mitochondrial structural damage and functional impairment (86)